Red Ginseng (RG) has been shown to improve insulin homeostasis and modulate cellular ageing processes in aged mice. It effectively reduces inflammation and cellular senescence in the liver, suggesting its potential as a beneficial supplement for ageing-related conditions.
December 2023 – MDPI
Key takeaways
- Enhanced insulin sensitivity and homeostasis: Red Ginseng improves insulin sensitivity and maintains insulin homeostasis in aged organisms. This action helps in delaying the onset of metabolic disorders that are commonly associated with ageing, such as type 2 diabetes and insulin resistance. By stabilising insulin levels, Red Ginseng supports metabolic health, which is crucial for longevity
- Reduction of inflammation and cellular senescence: The supplement significantly reduces inflammation and the presence of senescent cells in the liver. Inflammation and cellular senescence are key contributors to ageing and age-related diseases. By mitigating these factors, Red Ginseng acts to preserve the functionality of the liver, an organ critical to detoxification and metabolism, thereby potentially extending healthy lifespan
- Modulation of cellular pathways: Red Ginseng’s impact goes beyond simple symptom management; it actively modulates cellular pathways that are directly associated with the ageing process. This includes altering the expression of proteins involved in cell cycle arrest and apoptosis, suggesting a comprehensive approach to the prevention of cellular ageing and promotion of cellular rejuvenation
The study’s findings position Red Ginseng as a promising natural intervention for those seeking to mitigate the effects of ageing and pursue a longer, healthier life. Its ability to address multiple facets of the ageing process, from metabolic health to cellular integrity, underscores its potential as a supplement for enhancing overall longevity.
Read the article at: Kim, Yoo, et al. “Red Ginseng Attenuates the Hepatic Cellular Senescence in Aged Mice.” MDPI, vol. 13, no. 1, 8 Jan. 2024, https://www.mdpi.com/2079-7737/13/1/36.
