TERT activation and its potential to reverse ageing markers

A TERT activator was found to promote telomere synthesis, reduce cellular senescence, and alleviate neuroinflammation in aged mice, improving cognitive function. It achieved this by upregulating TERT transcription through the MEK/ERK/AP-1 pathway, reducing key ageing markers like p16 through DNA methylation, without increasing cancer risk.

Cellular senescence and its impact on metabolic ageing

Cellular senescence, where cells stop dividing but remain metabolically active, leads to the release of harmful molecules that drive ageing and metabolic dysfunction. Addressing the metabolic changes in senescent cells could help reduce ageing effects and related diseases.

POT1 and CST-Polα/primase protect telomeres and support healthy ageing

POT1 recruits and regulates the CST-Polα/primase complex at human telomeres, ensuring proper telomere replication and maintenance. This process is vital for maintaining genome stability, which plays a key role in aging, longevity, and preventing age-related diseases linked to telomere dysfunction.

MYCT1 gene discovery reveals key to blood stem cell renewal and longevity

MYCT1 is essential for how human hematopoietic stem cells (HSCs) sense and respond to their environment, which is crucial for their self-renewal and long-term function. This discovery points to potential therapies that could maintain healthy blood stem cells, support immune function, and enhance longevity by preserving stem cell regenerative abilities.

Rejuvenating aged stem cells to extend health and lifespan

Key strategies to rejuvenate aged stem cells and extend healthspan and lifespan include physical exercise, diet manipulation, targeting senescence, autophagy, epigenetic reprogramming, and using circulating blood factors. These methods enhance tissue homeostasis and regenerative capacity, potentially improving overall health and longevity.

Efficient DNA-protein crosslink repair enhances longevity

Endogenous aldehyde-induced DNA-protein crosslinks (DPCs) are efficiently resolved by transcription-coupled repair (TCR), protecting against metabolic genotoxins. This process explains the molecular pathogenesis of diseases like AMeDS and Cockayne syndrome. Effective aldehyde clearance and TCR are crucial for maintaining genomic integrity in transcribed regions.

The impact of genetics and lifestyle on brain ageing and longevity

Genetic and modifiable risk factors affect brain regions vulnerable to aging and diseases like Alzheimer's and schizophrenia. Key factors include diabetes, traffic-related air pollution, and alcohol intake. Modifying these risk factors could help protect these fragile...